沙門(mén)氏菌（L)多價(jià)抗原血清學(xué)鑒定

廣州健侖生物科技有限公司

我司長(zhǎng)期供應(yīng)尼古丁（可替寧）檢測(cè)試劑盒，違禁品檢測(cè)試劑盒，單卡檢測(cè)，3聯(lián)卡到12聯(lián)卡，可以自由組合，根據(jù)您的需求自由組合，*，性價(jià)比高，產(chǎn)品質(zhì)量很好。

保存要求：除了有特殊說(shuō)明，免疫檢測(cè)產(chǎn)品應(yīng)保存在2-8°C

產(chǎn)品規(guī)格：2ml/瓶

保質(zhì)期：2年

本試劑盒主要用于對(duì)病菌細(xì)菌進(jìn)行檢測(cè)，利用快速玻片凝集檢測(cè)技術(shù)

利用快速玻片凝集和對(duì)流免疫電泳(CIE)鑒定流感嗜血桿菌

沙門(mén)氏菌（L)多價(jià)抗原血清學(xué)鑒定

沙門(mén)氏菌（L)多價(jià)抗原血清學(xué)鑒定

【沙門(mén)氏知識(shí)點(diǎn)】

沙門(mén)氏等在霍亂流行時(shí)分離到豬霍亂沙門(mén)氏菌，故定名為沙門(mén)氏菌屬。沙門(mén)氏菌屬有的專對(duì)人類致病，有的只對(duì)動(dòng)物致病，也有對(duì)人和動(dòng)物都致病。沙門(mén)氏菌病是指由各種類型沙門(mén)氏菌所引起的對(duì)人類、家畜以及野生不同形式的總稱。感染沙門(mén)氏菌的人或帶菌者的糞便污染食品，可使人發(fā)生食物中毒。據(jù)統(tǒng)計(jì)在世界各國(guó)的種類細(xì)菌性食物中毒中，沙門(mén)氏菌引起的食物中毒常列。我國(guó)內(nèi)陸地區(qū)也以沙門(mén)氏菌為*。
沙門(mén)氏菌病的病原體。屬腸桿菌科，革蘭氏陰性腸道桿菌。已發(fā)現(xiàn)的近一千種（或菌株）。按其抗原成分，可分為甲、乙、丙、丁、戊等基本菌組。其中與人體疾病有關(guān)的主要有甲組的副傷寒甲桿菌，乙組的副傷寒乙桿菌和鼠傷寒桿菌，丙組的副傷寒丙桿菌和豬霍亂桿菌，丁組的傷寒桿菌和腸炎桿菌等。除傷寒桿菌、副傷寒甲桿菌和副傷寒乙桿菌引起人類的疾病外，大多數(shù)僅能目引起家畜、鼠類和禽類等動(dòng)物的疾病，但有時(shí)也可污染人類的食物而引起食物中毒。
沙門(mén)氏菌在水中不易繁殖，但可生存2-3周，冰箱中可生存3-4個(gè)月，在自然環(huán)境的糞便中可存活1-2個(gè)月。沙門(mén)氏菌zui適繁殖溫度為37℃，在20℃以上即能大量繁殖，因此，低溫儲(chǔ)存食品是一項(xiàng)重要預(yù)防措施。
沙門(mén)氏菌是一種常見(jiàn)的食源性致病菌。沙門(mén)氏菌鑒定的傳統(tǒng)方法主要是根據(jù)形態(tài)學(xué)特征、培養(yǎng)特征、生理生化特征、抗原特征、噬菌體特征等。

我司還有很多種血清學(xué)診斷血清、血液檢測(cè)、免疫檢測(cè)產(chǎn)品、毒素檢測(cè)、凝集檢測(cè)、酶免檢測(cè)、層析檢測(cè)、免疫熒光檢測(cè)產(chǎn)品，。

（ MOB：楊永漢）

我司還提供其它進(jìn)口或國(guó)產(chǎn)試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲(chóng)病、違禁品濫用、肺炎球菌、軍團(tuán)菌、化妝品檢測(cè)、食品安全檢測(cè)等試劑盒以及日本生研細(xì)菌分型診斷血清、德國(guó)SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。

想了解更多的產(chǎn)品及服務(wù)請(qǐng)掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司
【市場(chǎng)部】    楊永漢

【】 
【騰訊  】 
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103

休克時(shí)機(jī)體細(xì)胞內(nèi)溶酶體增多，體積增大，吞噬體顯著增加。溶酶體內(nèi)的酶向組織內(nèi)外釋放，多在肝和腸系膜等處，引起細(xì)胞和組織自溶。因此，在休克時(shí)，測(cè)定淋巴液和血液中溶酶體酶的含量高低，可作為細(xì)胞損傷輕重度的定量指標(biāo)。通常以酸性磷酸酶、β-葡萄糖醛酸酶與組織蛋白酶為指標(biāo)。關(guān)于休克時(shí)溶酶體釋放的機(jī)理，有人提出是由于H降低和三羧酸循環(huán)受阻。休克時(shí)缺血缺氧，引起細(xì)胞H值的下降(約H)，酸性水解酶活化，水解溶酶體膜，zui終導(dǎo)致溶酶體膜裂解，溶酶體釋放，使細(xì)胞、組織自溶。腫瘤溶酶體與腫瘤的關(guān)系日益引起人們的關(guān)注，一般有以下幾種觀點(diǎn)：()致癌物質(zhì)引起細(xì)胞分裂調(diào)節(jié)機(jī)能的障阻及染色體畸變，可能與溶酶體釋放水解酶的作用有關(guān)；()某些影響溶酶體膜通透性的物質(zhì)，如巴豆油，某些去垢劑、高壓氧等，是促進(jìn)致癌作用的輔助因子，也能引發(fā)細(xì)胞的異常分裂；()在核膜殘缺的情況下，核膜對(duì)核的保護(hù)喪失，溶酶體可以溶解染色質(zhì)，而引起細(xì)胞突變；()溶酶體代謝過(guò)程中的某些產(chǎn)物是腫瘤細(xì)胞增殖的物質(zhì)基礎(chǔ)；()致癌物質(zhì)進(jìn)入細(xì)胞。
During shock, lysosomes increased, the volume increased, and phagosome increased significantly. Lysosomal enzymes are released inside and outside the tissues, mostly in the liver and mesenteries, causing autolysis of cells and tissues. Therefore, during shock, the levels of lysosomal enzymes in lymph and blood can be measured and used as quantitative indicators of the severity of cell damage. Acid phosphatase, β-glucuronidase, and cathepsin are commonly used as indicators. The mechanism of lysosomal release during shock has been suggested to be due to a decrease in H and a block in the tricarboxylic acid cycle. Ischemia and hypoxia during shock cause a decrease in the H value of cells (about H), activation of acid hydrolyzing enzymes, hydrolysis of lysosomal membranes, eventually leading to lysosomal membrane breakdown, release of lysosomes, and autolysis of cells and tissues. The relationship between tumor lysosomes and tumors has increasingly attracted people's attention. There are generally the following viewpoints: () carcinogens cause cell division and regulation of functional barriers and chromosomal aberrations, may be related to the role of lysosomes to release hydrolytic enzymes; ( ) Some substances that affect the permeability of lysosomal membranes, such as croton oil, certain detergents, hyperbaric oxygen, etc., are cofactors that promote carcinogenesis and can also trigger abnormal cell division; () in nuclear membrane insufficiency In the case of nuclear membranes, the protection of the nucleus is lost, and lysosomes can dissolve chromatin and cause cell mutations; () Some of the products of lysosome metabolism are the material basis for the proliferation of tumor cells; () The entry of carcinogens cell

休克時(shí)機(jī)體細(xì)胞內(nèi)溶酶體增多，體積增大，吞噬體顯著增加。溶酶體內(nèi)的酶向組織內(nèi)外釋放，多在肝和腸系膜等處，引起細(xì)胞和組織自溶。因此，在休克時(shí)，測(cè)定淋巴液和血液中溶酶體酶的含量高低，可作為細(xì)胞損傷輕重度的定量指標(biāo)。通常以酸性磷酸酶、β-葡萄糖醛酸酶與組織蛋白酶為指標(biāo)。關(guān)于休克時(shí)溶酶體釋放的機(jī)理，有人提出是由于H降低和三羧酸循環(huán)受阻。休克時(shí)缺血缺氧，引起細(xì)胞H值的下降(約H)，酸性水解酶活化，水解溶酶體膜，zui終導(dǎo)致溶酶體膜裂解，溶酶體釋放，使細(xì)胞、組織自溶。腫瘤溶酶體與腫瘤的關(guān)系日益引起人們的關(guān)注，一般有以下幾種觀點(diǎn)：()致癌物質(zhì)引起細(xì)胞分裂調(diào)節(jié)機(jī)能的障阻及染色體畸變，可能與溶酶體釋放水解酶的作用有關(guān)；()某些影響溶酶體膜通透性的物質(zhì)，如巴豆油，某些去垢劑、高壓氧等，是促進(jìn)致癌作用的輔助因子，也能引發(fā)細(xì)胞的異常分裂；()在核膜殘缺的情況下，核膜對(duì)核的保護(hù)喪失，溶酶體可以溶解染色質(zhì)，而引起細(xì)胞突變；()溶酶體代謝過(guò)程中的某些產(chǎn)物是腫瘤細(xì)胞增殖的物質(zhì)基礎(chǔ)；()致癌物質(zhì)進(jìn)入細(xì)胞。
During shock, lysosomes increased, the volume increased, and phagosome increased significantly. Lysosomal enzymes are released inside and outside the tissues, mostly in the liver and mesenteries, causing autolysis of cells and tissues. Therefore, during shock, the levels of lysosomal enzymes in lymph and blood can be measured and used as quantitative indicators of the severity of cell damage. Acid phosphatase, β-glucuronidase, and cathepsin are commonly used as indicators. The mechanism of lysosomal release during shock has been suggested to be due to a decrease in H and a block in the tricarboxylic acid cycle. Ischemia and hypoxia during shock cause a decrease in the H value of cells (about H), activation of acid hydrolyzing enzymes, hydrolysis of lysosomal membranes, eventually leading to lysosomal membrane breakdown, release of lysosomes, and autolysis of cells and tissues. The relationship between tumor lysosomes and tumors has increasingly attracted people's attention. There are generally the following viewpoints: () carcinogens cause cell division and regulation of functional barriers and chromosomal aberrations, may be related to the role of lysosomes to release hydrolytic enzymes; ( ) Some substances that affect the permeability of lysosomal membranes, such as croton oil, certain detergents, hyperbaric oxygen, etc., are cofactors that promote carcinogenesis and can also trigger abnormal cell division; () in nuclear membrane insufficiency In the case of nuclear membranes, the protection of the nucleus is lost, and lysosomes can dissolve chromatin and cause cell mutations; () Some of the products of lysosome metabolism are the material basis for the proliferation of tumor cells; () The entry of carcinogens cell

休克時(shí)機(jī)體細(xì)胞內(nèi)溶酶體增多，體積增大，吞噬體顯著增加。溶酶體內(nèi)的酶向組織內(nèi)外釋放，多在肝和腸系膜等處，引起細(xì)胞和組織自溶。因此，在休克時(shí)，測(cè)定淋巴液和血液中溶酶體酶的含量高低，可作為細(xì)胞損傷輕重度的定量指標(biāo)。通常以酸性磷酸酶、β-葡萄糖醛酸酶與組織蛋白酶為指標(biāo)。關(guān)于休克時(shí)溶酶體釋放的機(jī)理，有人提出是由于H降低和三羧酸循環(huán)受阻。休克時(shí)缺血缺氧，引起細(xì)胞H值的下降(約H)，酸性水解酶活化，水解溶酶體膜，zui終導(dǎo)致溶酶體膜裂解，溶酶體釋放，使細(xì)胞、組織自溶。腫瘤溶酶體與腫瘤的關(guān)系日益引起人們的關(guān)注，一般有以下幾種觀點(diǎn)：()致癌物質(zhì)引起細(xì)胞分裂調(diào)節(jié)機(jī)能的障阻及染色體畸變，可能與溶酶體釋放水解酶的作用有關(guān)；()某些影響溶酶體膜通透性的物質(zhì)，如巴豆油，某些去垢劑、高壓氧等，是促進(jìn)致癌作用的輔助因子，也能引發(fā)細(xì)胞的異常分裂；()在核膜殘缺的情況下，核膜對(duì)核的保護(hù)喪失，溶酶體可以溶解染色質(zhì)，而引起細(xì)胞突變；()溶酶體代謝過(guò)程中的某些產(chǎn)物是腫瘤細(xì)胞增殖的物質(zhì)基礎(chǔ)；()致癌物質(zhì)進(jìn)入細(xì)胞。
During shock, lysosomes increased, the volume increased, and phagosome increased significantly. Lysosomal enzymes are released inside and outside the tissues, mostly in the liver and mesenteries, causing autolysis of cells and tissues. Therefore, during shock, the levels of lysosomal enzymes in lymph and blood can be measured and used as quantitative indicators of the severity of cell damage. Acid phosphatase, β-glucuronidase, and cathepsin are commonly used as indicators. The mechanism of lysosomal release during shock has been suggested to be due to a decrease in H and a block in the tricarboxylic acid cycle. Ischemia and hypoxia during shock cause a decrease in the H value of cells (about H), activation of acid hydrolyzing enzymes, hydrolysis of lysosomal membranes, eventually leading to lysosomal membrane breakdown, release of lysosomes, and autolysis of cells and tissues. The relationship between tumor lysosomes and tumors has increasingly attracted people's attention. There are generally the following viewpoints: () carcinogens cause cell division and regulation of functional barriers and chromosomal aberrations, may be related to the role of lysosomes to release hydrolytic enzymes; ( ) Some substances that affect the permeability of lysosomal membranes, such as croton oil, certain detergents, hyperbaric oxygen, etc., are cofactors that promote carcinogenesis and can also trigger abnormal cell division; () in nuclear membrane insufficiency In the case of nuclear membranes, the protection of the nucleus is lost, and lysosomes can dissolve chromatin and cause cell mutations; () Some of the products of lysosome metabolism are the material basis for the proliferation of tumor cells; () The entry of carcinogens cell

休克時(shí)機(jī)體細(xì)胞內(nèi)溶酶體增多，體積增大，吞噬體顯著增加。溶酶體內(nèi)的酶向組織內(nèi)外釋放，多在肝和腸系膜等處，引起細(xì)胞和組織自溶。因此，在休克時(shí)，測(cè)定淋巴液和血液中溶酶體酶的含量高低，可作為細(xì)胞損傷輕重度的定量指標(biāo)。通常以酸性磷酸酶、β-葡萄糖醛酸酶與組織蛋白酶為指標(biāo)。關(guān)于休克時(shí)溶酶體釋放的機(jī)理，有人提出是由于H降低和三羧酸循環(huán)受阻。休克時(shí)缺血缺氧，引起細(xì)胞H值的下降(約H)，酸性水解酶活化，水解溶酶體膜，zui終導(dǎo)致溶酶體膜裂解，溶酶體釋放，使細(xì)胞、組織自溶。腫瘤溶酶體與腫瘤的關(guān)系日益引起人們的關(guān)注，一般有以下幾種觀點(diǎn)：()致癌物質(zhì)引起細(xì)胞分裂調(diào)節(jié)機(jī)能的障阻及染色體畸變，可能與溶酶體釋放水解酶的作用有關(guān)；()某些影響溶酶體膜通透性的物質(zhì)，如巴豆油，某些去垢劑、高壓氧等，是促進(jìn)致癌作用的輔助因子，也能引發(fā)細(xì)胞的異常分裂；()在核膜殘缺的情況下，核膜對(duì)核的保護(hù)喪失，溶酶體可以溶解染色質(zhì)，而引起細(xì)胞突變；()溶酶體代謝過(guò)程中的某些產(chǎn)物是腫瘤細(xì)胞增殖的物質(zhì)基礎(chǔ)；()致癌物質(zhì)進(jìn)入細(xì)胞。
During shock, lysosomes increased, the volume increased, and phagosome increased significantly. Lysosomal enzymes are released inside and outside the tissues, mostly in the liver and mesenteries, causing autolysis of cells and tissues. Therefore, during shock, the levels of lysosomal enzymes in lymph and blood can be measured and used as quantitative indicators of the severity of cell damage. Acid phosphatase, β-glucuronidase, and cathepsin are commonly used as indicators. The mechanism of lysosomal release during shock has been suggested to be due to a decrease in H and a block in the tricarboxylic acid cycle. Ischemia and hypoxia during shock cause a decrease in the H value of cells (about H), activation of acid hydrolyzing enzymes, hydrolysis of lysosomal membranes, eventually leading to lysosomal membrane breakdown, release of lysosomes, and autolysis of cells and tissues. The relationship between tumor lysosomes and tumors has increasingly attracted people's attention. There are generally the following viewpoints: () carcinogens cause cell division and regulation of functional barriers and chromosomal aberrations, may be related to the role of lysosomes to release hydrolytic enzymes; ( ) Some substances that affect the permeability of lysosomal membranes, such as croton oil, certain detergents, hyperbaric oxygen, etc., are cofactors that promote carcinogenesis and can also trigger abnormal cell division; () in nuclear membrane insufficiency In the case of nuclear membranes, the protection of the nucleus is lost, and lysosomes can dissolve chromatin and cause cell mutations; () Some of the products of lysosome metabolism are the material basis for the proliferation of tumor cells; () The entry of carcinogens cell